The reverse stop-signal model can potentially explain how CTLA4 regulates the activation threshold of T cells, anergy, autoimmunity, tissue infiltration and various T-cell effector functions.

A decomposition formula for the implied skew of a local stochastic volatility model suggests this can be achieved via a non-smooth leverage function, such as a step function.

MathWorks and SiSoft showcase new solutions and methodology to capture analog and digital effects at the system-level.